<oai_dc:dc xmlns:dc="http://purl.org/dc/elements/1.1/" xmlns:oai_dc="http://www.openarchives.org/OAI/2.0/oai_dc/" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance" xsi:schemaLocation="http://www.openarchives.org/OAI/2.0/oai_dc/ http://www.openarchives.org/OAI/2.0/oai_dc.xsd">
  <dc:creator>Guerra, Jessica</dc:creator>
  <dc:creator>Guarda, Greta</dc:creator>
  <dc:date>2024</dc:date>
  <dc:description xmlns:ns0="xml" ns0:lang="en">NLRC5 is a transcriptional regulator of genes governing T cell responses. Most characterized NLRs are instead innate immune sensors forming complexes leading to pyroptosis. Raising exciting questions, Sundaram and colleagues now demonstrate that NLRC5 forms large complexes and causes PANoptosis (immunogenic cell death), in response to heme in inflammatory contexts.</dc:description>
  <dc:format>application/pdf</dc:format>
  <dc:identifier>https://n2t.net/ark:/12658/srd1331167</dc:identifier>
  <dc:identifier>https://susi.usi.ch/global/documents/331167</dc:identifier>
  <dc:identifier>https://susi.usi.ch/documents/331167/files/Guarda_2024_Cell Press_Trends immun_NLRC5 back to innate immunity.pdf</dc:identifier>
  <dc:language>eng</dc:language>
  <dc:relation>info:eu-repo/semantics/altIdentifier/doi/10.1016/j.it.2024.07.002</dc:relation>
  <dc:relation>info:eu-repo/semantics/altIdentifier/ark/12658/srd1331167</dc:relation>
  <dc:rights>info:eu-repo/semantics/openAccess</dc:rights>
  <dc:rights>CC BY-NC-ND</dc:rights>
  <dc:source>Trends in immunology . - 2024</dc:source>
  <dc:subject>info:eu-repo/classification/udc/61</dc:subject>
  <dc:title xmlns:ns1="xml" ns1:lang="en">NLRC5 : back to innate immunity</dc:title>
  <dc:type>http://purl.org/coar/resource_type/c_6501</dc:type>
</oai_dc:dc>